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ninety two demonstrated that improved preload may end in enhanced myocyte Ca2+-entry secondary to mechanical pressure independent of ischaemia, thus resulting in activation of μ-calpains and calpain-mediated cTnI proteolysis.

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murine knockout models. cTnT-knockout results in sarcomere disassembly and early embryonic lethality.18 Remarkably, knockout of cTnI in murine embryos has no effect on well being before postnatal day 15, attributable to ssTnI compensating for The dearth of cTnI.

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21 respectively. Identification on the AA sequences for human cTnI and cTnT followed and were first reported by Vallins et al.

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